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    Arrhythmia research - key publications using PLN antibodies

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    References

     

    1. Ai, X. et al. 2005. Ca2+/calmodulin-dependent protein kinase modulates cardiac ryanodine receptor phosphorylation and sarcoplasmic reticulum Ca2+ leak in heart failure. Circulation Research. 97(12), pp.1314-1322.
    2. Andre, L. et al. 2011. Carbon monoxide exposure enhances arrhythmia after cardiac stress: involvement of oxidative stress. Basic Res Cardiol. 106(6), pp.1235-46.
    3. Bovo, E. et al. 2012. Reactive oxygen species contribute to the development of arrhythmogenic Ca(2)(+) waves during beta-adrenergic receptor stimulation in rabbit cardiomyocytes. J Physiol. 590(Pt 14), pp.3291-304.
    4. Dybkova, N. et al. 2011. Overexpression of CaMKII delta c in RyR2(R4496C+/-) Knock-In Mice Leads to Altered Intracellular Ca2+ Handling and Increased Mortality. Journal of the American College of Cardiology. 57(4), pp.469-479.
    5. El-Armouche, A. et al. 2011. Phospholemman-dependent regulation of the cardiac Na/K-ATPase activity is modulated by inhibitor-1 sensitive type-1 phosphatase. Faseb Journal. 25(12), pp.4467-4475.
    6. Freestone, N.S. et al. 1999. beta(4)-adrenoceptors are more effective than beta(1)-adrenoceptors in mediating arrhythmic Ca2+ transients in mouse ventricular myocytes. Naunyn-Schmiedebergs Archives of Pharmacology. 360(4), pp.445-456.
    7. Gergs, U. et al. 2010. Cardiac overexpression of the human 5-HT4 receptor in mice. American Journal of Physiology-Heart and Circulatory Physiology. 299(3), pp.H788-H798.
    8. Goette, A. et al. 2012. The impact of rapid atrial pacing on ADMA and endothelial NOS. Int J Cardiol. 154(2), pp.141-6.
    9. Greiser, M. et al. 2009. Distinct contractile and molecular differences between two goat models of atrial dysfunction: AV block-induced atrial dilatation and atrial fibrillation. Journal of Molecular and Cellular Cardiology. 46(3), pp.385-394.
    10. Kalyanasundaram, A. et al. 2012. Functional consequences of stably expressing a mutant calsequestrin (CASQ2(D307H)) in the CASQ2 null background. American Journal of Physiology-Heart and Circulatory Physiology. 302(1), pp.H253-H261.
    11. Qi, X. et al. 2009. The Calcium/Calmodulin/Kinase System and Arrhythmogenic Afterdepolarizations in Bradycardia-Related Acquired Long-QT Syndrome. Circulation-Arrhythmia and Electrophysiology. 2(3), pp.295-304.
    12. Ruan, H.M. et al. 2007. Gi alpha 1-mediated cardiac electrophysiological remodeling and arrhythmia in hypertrophic cardiomyopathy. Circulation. 116(6), pp.596-605.
    13. Ruiz-Hurtado, G. et al. 2012a. Cardiotrophin-1 induces sarcoplasmic reticulum Ca-2 leak and arrhythmogenesis in adult rat ventricular myocytes. Cardiovascular Research. 96(1), pp.81-89.
    14. Said, M. et al. 2008. Increased intracellular Ca2+ and SR Ca2+ load contribute to arrhythmias after acidosis in rat heart. Role of Ca2+/calmodulin-dependent protein kinase II. American Journal of Physiology-Heart and Circulatory Physiology. 295(4), pp.H1669-H1683.
    15. Song, L. et al. 2007. Calsequestrin 2 (CASQ2) mutations increase expression of calreticulin and ryanodine receptors, causing catecholaminergic polymorphic ventricular tachycardia. Journal of Clinical Investigation. 117(7), pp.1814-1823.
    16. Thireau, J. et al. 2014. Beta-adrenergic blockade combined with subcutaneous B-type natriuretic peptide: a promising approach to reduce ventricular arrhythmia in heart failure? Heart. 100(11), pp.833-41.
    17. Tsao, H.M. et al. 2012. Amyloid peptide regulates calcium homoeostasis and arrhythmogenesis in pulmonary vein cardiomyocytes. European Journal of Clinical Investigation. 42(6), pp.589-598.
    18. Yajima, T. et al. 2011. Absence of SOCS3 in the Cardiomyocyte Increases Mortality in a gp130-Dependent Manner Accompanied by Contractile Dysfunction and Ventricular Arrhythmias. Circulation. 124(24), pp.2690-U262.

     

     

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